Cholesterol metabolites alleviate injured liver function and decrease mortality in an LPS-induced mouse model
Sulfation of 25‑hydroxycholesterol regulates lipid metabolism, inflammatory responses, and cell proliferation
Oxysterol sulfation by cytosolic sulfotransferase suppresses liver X receptor/sterol regulatory element binding protein‑1c signaling pathway and reduces serum and hepatic lipids in mouse models of nonalcoholic fatty liver disease
